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I think thats low-hanging fruit that we could really get better at, in terms of rehab

The hypothesized mechanism for this therapeutic effect is that the increased activity of GABAergic neurons, induced by the NMDAR agonist, prevents the pathological increase in gain of auditory cortex, thus preventing the development of tinnitus

The other carbons are the same as in the previous molecule but are numbered one less than in the reactant (because CO2 was release)

doi: 10.1074/jbc.M405127200

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