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This stress leads to suppression of forkhead box O1 (FoxO1) signaling and subsequent upregulation of thioredoxin interacting protein, inhibition of insulin and SOD-2 expression, re-expression of Neurog3, and -cell dedifferentiation and functional failure

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Selectively interacts with ERR subtypes for examining mitochondrial biology and metabolic regulation.

Glutathione is particularly important in detoxification pathways, especially in the liver, where it facilitates the breakdown and removal of metabolic by-products, toxins, and heavy metals

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