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The absence of serious adverse events across varied doses, administration routes, and treatment durations represents a key safety finding that distinguishes AOD-9604s clinical profile

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A peer-reviewed study in the Journal of Endocrinology confirming that AOD9604 produces lipolysis and increases fat oxidation in obese mice via the beta-adrenergic pathway, with effects comparable to full-length HGH but without the insulin-disrupting side effects provided early mechanistic validation that informed the human trials

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