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These changes shift energy metabolism from glycolysis toward OXPHOS, increase ATP levels and mitochondrial ROS, and activate p53 via ATR-dependent stress signaling

Cancer 19 , 405414 (2019)

oatesi , L

Metabolomic approaches have identified unique profiles in NASH patients, with specific metabolites like ethanol linked to disease progression

Type 2 diabetes mellitus and the risk of abnormal spermatozoa: A Mendelian randomization study

Our data are consistent with a model in which loss of unc1 function perturbs gap junctions between IL2 and ASJ, resulting in altered signalling from ASJ
