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In addition to NAFLD, NASH is also of concern, with limited treatment options
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Insulin resistance in skeletal muscle, liver, and adipocytes (1115) and -cell dysfunction (1622) remain the major core defects responsible for the development and progression of hyperglycemia
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Likewise, daily liraglutide treatment downregulated lung C-C motif chemokine ligand 2 (Ccl2) and IL-6 in mice during influenza infection
These are typically dose dependent, occur most frequently upon initiation or dose escalation, and often diminish over time [84]
