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apoE / mice were treated with low- or high dose exendin-4GLP-1 receptor agonist ( ) and MCP-1inflammatory and atherogenic mediators

Thus, combining GIPR and GLP-1R agonism may enhance insulin secretion in patients with T2D by first rescuing dysfunctional beta-cells with GLP-1R agonism followed by additive effects on insulin secretion provided by both receptors

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Tham LS, Schneck KB, Geiser JS, Posada M, Dickinson G