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To validate a structural change in tobacco smoke-condensate-treated endothelial cells, we performed SA--gal staining and quantified the protein expression of Lamin B1 by doing IF staining and WB

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In fact, p53 accumulation (due to increased half-life of the protein) was detected in UVR-exposed skin cells, where p53 plays an active function as a transcriptional regulator [11]

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Repression of death receptor-mediated apoptosis of hepatocytes by hepatitis B virus E antigen

Cholinergic systems in brain development and disruption by neurotoxicants: nicotine, environmental tobacco smoke, organophosphates