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In experimental models of chronic inflammation, animals treated with GHK-Cu show 50% faster resolution of inflammatory lesions and 70% reduced tissue damage compared to untreated controls

doi: 10.1076/soph.17.3.206.14784 17 LemmensSHuaMTStalmansI

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In addition, we have mentioned the protective role of mitochondrial ALDH2 against oxidative stress-mediated mitochondrial dysfunction and hepatotoxicity, apoptosis/necrosis of hepatocytes and gut enterocytes, the production of DAMPs, the activation of Kupffer cells and HSCs, altered inflammation, and fibrosis, leading to cirrhosis and HCC

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Vitamin E as an antioxidant/free radical scavenger against amyloid beta-peptide-induced oxidative stress in neocortical synaptosomal membranes and hippocampal neurons in culture: insights into Alzheimers disease