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Dear editor A recent study published in Annals of the Rheumatic Diseases suggests that the expression of hypoxia-inducible factor 1 alpha (HIF1a) in intestinal epithelial cells (IECs) represents a critical check point for the development of arthritis by inhibiting necroptosis of IECs and preserving the intestinal barrier integrity [1]

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We detected GLUT1 upregulation of the CMV-specific CD38 hi compared with CD38 lo CD8 + T cells in controllers and noncontrollers (Figure 5, F and G)

Recordings were performed in an isolation chamber to prevent the fish from responding to other cues