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Targeting c-MET in gastrointestinal tumours: rationale, opportunities and challenges

Nat Mater

Andersen, S

In addition, the classical ferroptosis inducer erastin was also reported to damage cardiomyocytes by initiating ferroptosis, while ENPP2, a lipid kinase generating the lipid mediator lysophosphatidic acid, could protect cardiomyocytes from the erastin-induced ferroptosis by decreasing ROS generation [107]

3 The gastrointestinal side-effect profile seen across its trials is the clinical fingerprint of that mechanism

A rate of increase of 1 fluorescence unit per minute on the recorder was calibrated to correspond to a rate of formation of conjugate of 0.1 nmoles ml -1 min -1