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doi: 10.3168/jds.2021-21483 57

The substrates and products of the DHODH reaction have opposite effects on ferroptosis, suggesting that the potential role of DHODH in regulating ferroptosis sensitivity is independent of its function of producing pyrimidine nucleotides

206 Chen et al

10.1038/s41467-018-05720-5 47 NaY

(1988), published in FEBS Letters , which found that GHK-Cu exposure at nanomolar concentrations in primary fibroblast cultures produced measurable increases in collagen synthesis relative to untreated controls.[5] This established the peptide's basic identity as a fibroblast-activating signal and prompted the wave of ECM-focused research that followed