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Description
Gut microbes promote colonic serotonin production through an effect of short-chain fatty acids on enterochromaffin cells

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Its main roles are the stimulation of insulin release [30], suppression of glucagon secretion [31], delay of gastric emptying and reduction in food intake [32]

The observed increase in ROS/RNS in GLT-1 cKO appears to manifest within postsynaptic neurons rather than astrocytes, as evidenced by the unchanged activity of GS

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