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Cur@SF NPs alleviate Friedreich's ataxia in a mouse model through synergistic iron chelation and antioxidation

In vitro, we could demonstrate that the ENL YEATS domain interacts with CRBNDDB1 (damage-specific DNA-binding protein 1) and the newly reported CRBN midi construct 41 but only in the presence of the compound (Fig

Additionally, other pathways such as the Hodge, Wolff, and acetyl alcohol pathways also contribute to AGE formation, often involving free radicals and metal ions

The exact mechanisms by which ROS control memory formation or function are not well understood