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Clinical Reviews in Gastroenterology, 23(2), 89-102

Many cardiovascular risk factors such as diabetes mellitus, hypertension and hyperlipidemia are demonstrated to stimulate the interaction of Cav-1 and eNOS, reduce nitric oxide production resulting in endothelial dysfunction and subsequently the formation of atherosclerotic lesion 41,42,43

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Research literature characterised honestly qualified throughout ("research models," "in vitro/in vivo/clinical" distinction made explicit), no overstatement