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It establishes cellular antioxidant defense by transcriptionally activating antioxidant genes (e.g., SOD2, CAT, GCL) and reductive molecules (e.g., GSH, Trx), while downregulating pro-oxidant molecule synthesis, thereby directly scavenging or neutralizing excess ROS [134]

& Pervaiz, S

In chemotherapy-sensitive patients, Keap1 (Kelch-like ECH-associated protein 1) binds to NRF2 and mediates NRF2 ubiquitination via the Cul3-E3 ligase complex, promoting its degradation and maintaining NRF2 at low levels [32]

Neuropathy target esterase (NTE/PNPLA6) and organophosphorus compound-induced delayed neurotoxicity (OPIDN)

[DOI] [PubMed] 10.Schalka S, dos Reis VM, Cuce LC

doi: 10.1038/s41565-022-01266-2 147 LuoZHuangYBatraNChenYHuangHWangYet al