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2), and the immunomodulatory activities of each compound are presented in Table 3

Friedreichs ataxia (FRDA) is a neurodegenerative disease caused by a defect in mitochondrial frataxin (FXN), a key regulator of ferroptosis, and patients with FRDA are predisposed to diabetes (80)

Overexpression of miR-126 could reverse the decrease in AKT and eNOS phosphorylation caused by high glucose, exerting a protective effect on anti-inflammatory symptoms [93]

D., Joo, H., Mosberg, H

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425,426,427,428 Targeting the Keap1/PGAM5/AIFM1 axis in ROS-mediated oxieptosis could selectively amplify oxidative stress to induce programmed cell death in cancer cells, offering a novel therapeutic strategy