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In comparison, WT cells showed only minor, temperaturedependent changes in fatty acid composition caused by homeoviscous adaptation towards increased SFA content at higher temperatures (Fig 1E and Appendix Fig S2C)

This so-called autocrine loop of ROS by HSCs is regarded as mechanism corresponding to the autocrine loop of TGF- which HSCs produce in response to this cytokine with an increased collagen expression in the injured liver (Itagaki et al., 2005)