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In the APP/PS1 mouse model of AD, administration of AKG or calcium -ketoglutarate (Ca-AKG) ameliorated impaired long-term potentiation at hippocampal CA1 synapses, with more pronounced effects observed in female AD mice

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An arterial or venous sample can be used

Normal levels of intracellular GSH may exert a critical negative control on the elaboration of proinflammatory cytokines

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