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This study revealed a novel mechanism through which TSA regulates galectin-3 expression via ALKBH5-mediated m6A modification, providing a potential therapeutic target for cardiac hypertrophy (Fig
Although the peptide might not be completely ruined, the integrity of the solution may be compromised, leading to inconsistent results

[Abstract]2025 Jun 14:19:5085-5098

10.1073/pnas.1721469115

To view a copy of this licence, visit About this article Cite this article Sawami, K., Tanaka, A

Shake out 1 test strip and close the bottle
