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Fatty acid labeling from glutamine in hypoxia can be explained by isotope exchange without net reductive isocitrate dehydrogenase (IDH) flux

However, it may be limited in its definition of gout, which was diagnosed based on clinical criteria rather than joint aspiration, suggesting that some enrolled patients may not have had gout

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(1988), published in FEBS Letters , which found that GHK-Cu exposure at nanomolar concentrations in primary fibroblast cultures produced measurable increases in collagen synthesis relative to untreated controls.[5] This established the peptide's basic identity as a fibroblast-activating signal and prompted the wave of ECM-focused research that followed
