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GSH, however, is susceptible to oxidation over time in a phosphate buffer environment

Using selected doses and time point for ASA toxicity, we have demonstrated that when GSH synthesis is inhibited in HepG2 cells by buthionine sulfoximine (BSO), prior to ASA treatment, cytotoxicity of the drug is augmented

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Conclusions It is concluded that ED drugs induced the activities of both SOD and catalase which consequently decreased MDA level