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Description
Illustratively for the counteraction of the isoprenaline myocardial infarction as well, the BPC 157 activity as a stabilizer of cellular junction (counteracting leaky gut syndrome) occurs via increasing tight junction protein ZO-1 expression and transepithelial resistance [30]

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BPC-157 activates the VEGFR2-Akt-eNOS pathway, increasing blood vessel formation by 129% to 152% in damaged tissues according to preclinical studies
