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Subsequent studies demonstrated that exogenous administration of thymosin beta-4 promoted corneal wound healing, dermal repair, and cardiac tissue recovery in rodent models

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As an underlying mechanism, genistein appears to inhibit oxidative stress-stimulated c-Src kinase activation, preventing the tyrosine phosphorylation of TJ proteins (ZO-1 and occludin) and adherence junction (AJ) proteins (E-cadherin)
