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The 5xFAD transgenic mouse line has been genetically engineered to model features of AD through the expression of human amyloid precursor protein (APP) and presenilin-1 transgenes, which are associated with early-onset AD (Brody et al., 2017)
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It counteracts the behavioral and neurochemical effects of both dopamine agonists (amphetamine) and antagonists (haloperidol)
As shown in Figures 6AD, DSS-induced mice exhibited significantly increased SIRT1 expression and decreased p-STAT3 expression compared to the Control group
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