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The EBV latency III induces resistance to ferroptosis inducing agents To gain insights into how EBV latency programs alter lipid ROS levels and ferroptosis vulnerability, we utilized isogenic Burkitt tumor derived MUTU I vs III cell lines, which differ only by EBV latency I vs III programs, (43)

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44 and 46 sometimes showed higher intracellular uptake into Hela cells than 41 , nevertheless, the uptake of 45 was strongly limited

According to Grant [18] an exposure to various environmental factors (selenium) causing oxidative stress leads to an increase in the concentration of glutathione in the cells

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