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These molecules then enter the Krebs cycle in the mitochondria, ultimately producing energy in the form of ATP

het regelt het transport van vetzuren naar de mitochondrin (1, 2, 3)

For laboratory and in-vitro research use only

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First, by enhancing fatty acid transport into the mitochondria, it theoretically ensures that a greater proportion of the energy deficit is filled by fat oxidation rather than muscle protein breakdown

however, this progresses to channel inhibition within 3 min of incubation (Liu and Rosenberg, 1996)