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On the other hand, a low ROS state shows an OXPHOS-dominated M2 metabolic mode, featured by efficient ETC and reduced ROS production, which is conducive to the maintenance of M2 polarization and facilitates anti-inflammatory and repair functions (90)

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inhibition of the p16 INK4a /retinoblastoma (Rb) pathway, preventing phosphorylation of Rb protein and maintaining cell cycle arrest

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