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2 AdedoyinO.BodduR.TraylorA.LeverJ
From a mechanistic perspective, these outcomes are mediated by multi-system endocrine adaptations, including suppression of the HPG axis, alterations in insulin and glucose-regulating hormones, reduced triiodothyronine (T3), elevated cortisol, and disruptions in growth hormone (GH) and insulin-like growth factor 1 (IGF-1) signaling [92]
The prognostic value of serum amyloid A in solid tumors: A meta-analysis
doi:10.7326/0003-4819-143-3-200508020-00006
Crit Rev Toxicol 42:230243
As the neural progenitor cell population becomes dysfunctional with age MGF is not able to induce mitogenic effects
