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In hyperglycemic conditions, MSCs suppress pro-inflammatory cytokines through NF-B inhibition and enhance antioxidant defenses by activating the nuclear factor erythroid 2-related factor 2 (Nrf2) pathway

[DOI] [PMC free article] [PubMed] [Google Scholar] Raj K

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Delorge I, Figueroa CM, Feil R, Lunn JE, Van Dijck P

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Several key pathways including Notch, phosphatidylinositol 3-kinase/protein kinase B (PI3K/Akt), transforming growth factor- (TGF-), nuclear factor kappa (NF-B), bone morphogenetic protein (BMP), N-methyl-D-aspartic acid receptor (NMDAR)-Ca 2+ -Activin A, hippo-yes-associated protein (YAP), toll-like receptor 4/toll-interleukine-1 receptor domain-containing adapter-inducing interferon- (TLR4/TRIF) and hepatocyte growth factor/mesenchymal-epithelial transition factor (HGF/c-Met), and Wnt, have emerged as crucial players in this context