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It has been observed that the apoptotic process in paclitaxel induced-painful neuropathy is due to the permeability transition pore opening, with the consequent cytochrome c release and Ca 2+ homeostasis dysregulation (Areti et al., in vivo could also reverse or attenuate the above-mentioned symptoms, but with deleterious effects on motor coordination in the case of complex I inhibition at 3 and 24 h after paclitaxel administration

Am J Hum Genet 2001;69:88-95

Nuclear factor-B (NF-B) stimulates several pro-inflammatory factors that activate COX-2 and inducible nitric oxide synthase (iNOS) [53]

[Zugriff 5.5.20.] Hagen TM et al

Therefore, these aforementioned studies suggest that the RNA m 6 A modification is involved in ferroptosis by regulating the mRNA stability and protein expression of multiple ferroptosis-related genes (Figure 4B)