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However, the source of free GA in human metabolism is not fully understood since interrupting the saccharopine pathway upstream of glutaryl-CoA formation in a mouse model for GA-I did not rescue the mice from developing the disease 42

Nicotinamide nucleotide transhydrogenase regulates mitochondrial metabolism in NSCLC through maintenance of FeS protein function

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Its potential anticancer effects may be linked to the activation of the NRF2-ARE signaling pathway and its associated cytoprotective properties [155]