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In the absence of oxygen, there is little mitochondrial NAD + available for the pyruvate dehydrogenase complex, thus no acetyl-CoA will be formed, pyruvate carboxylase will not get activated, there will be no citrate, there will be no citric acid cycle, ATP levels will be low, thus phosphofructokinase-1 will remain uninhibited (because of no citrate and low [ATP] accompanied by high [ADP])

[DOI] [PubMed] [Google Scholar] 24.Prusinski Fernung L.E., Al-Hendy A., Yang Q

Recently, other ferroptosis protective mechanisms such as FSP1 [20, 56] and GCH1 [19] have been discovered, which often compensate for GPX4 reduction

Weintraub S

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(PubMed) Kennedy DO, Veasey R, Watson A, et al