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Increased NFB activation in atherosclerosis susceptible areas leads to endothelial cell activation (Figure 1), as evidenced by increased expression of monocyte adherence proteins (VCAM-1, ICAM-1, and P-selectin) and proinflammatory receptors (toll-like receptor 2, TLR2) and cytokines (MCP-1 and IL-8) (19,23,24)

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Also, Apo M/S1P may activate PI3K/Akt pathway by S1PR1 and S1PR3 to irritate eNOS expression and NO generation, lessening monocytes adhesion to ECs 95,96

2019;38 doi: 10.1186/s13046-019-1461-z.464 [DOI] [PMC free article] [PubMed] [Google Scholar] 99.Baci D., Bruno A., Bassani B., et al