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Loss- and gain-of-function experiments demonstrated that PARN knockdown attenuated the MAT2A-induced promotion of proliferation and migration, while reversing its inhibitory effect on apoptosis in MAT2A-overexpressing OS cells (Fig

6f), whereas Ru-hydroxide treatment significantly attenuates this signal to levels comparable to Ctrl conditions, as quantitatively demonstrated in Fig

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Nephron 56 , 5055 (1990)

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