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doi: 10.1034/j.1399-5618.2000.020302.x

Mechanistically, DAAs inhibit specific HCV non-structural proteins (NS) that are vital for its replication

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Therapy-resistant melanomas often undergo de-differentiation, which is characterized by loss of melanocytic markers such as MLANA, TRPM1 and TYR and gain of invasive molecular markers such as c-JUN, NGFR and ZEB1 ( Figure 1A )