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Although ACSL4 is consistently upregulated in OA cartilage and linked to ferroptosis progression, its inhibition must be balanced to avoid disrupting normal PUFA metabolism required for membrane integrity and repair (74, 77)

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Bei vielen chronischen Erkrankungen sinkt der Glutathionspiegel Auch bei chronischen Erkrankungen sinkt der Spiegel

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Significant immune reactivity towards all of these markers of oxidative damage in the white matter, which often extended well into the grey matter of axons, was observed in every case of autism examined

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