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Palmitoylation of cysteine residues located at the N-terminus of GAD65 is required for the transport of GAD65 to synaptic terminals [51], and decreased palmitoylation impairs GABAergic neurotransmission, as observed in Huntingtons disease [52]

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In vivo, subcutaneous xenograft models were established by injecting MNNG/HOS cells with or without MAT2A knockdown

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Published research/clinical protocols have reported daily dosing ranges such as