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Importantly, these metabolic changes were not reversed by LOS, possibly because competitive antagonism may not fully block AGTR1 under supraphysiological AngII concentrations, leaving residual receptor activity that could sustain bioenergetic adjustments through mechanisms, such as Ca 2+ -linked events, that are probably pharmacologically less sensitive and functionally buffered by the high basal metabolic tone of T24 cells

Their exceptional chemical stability allows environmental persistence exceeding two decades, leading to progressive bioaccumulation through food webs and preferential storage in lipid-rich tissues ( Human exposure to these compounds occurs through multiple pathwaysingestion, inhalation, and dermal absorptionwith subsequent accumulation in adipose tissue, hepatic systems, and critically, reproductive organs ( 1.2 Key research challenges Despite substantial advances in understanding individual EED toxicity profiles, several fundamental knowledge gaps continue to impede comprehensive risk assessment: Complex Mixture Effects Remain Poorly Understood While toxicological studies have extensively characterized individual EED effects, real-world human exposure invariably involves simultaneous contact with multiple compounds