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This cascade contributes to ovarian dysfunction in PCOS by interfering with follicular development, insulin signaling, and inflammation [21,31,32], highlighting the critical need to maintain redox balance for reproductive and metabolic health

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Ahrens, C

In contrast, the CAT and GSH contents were significantly increased by 20% and 21%, respectively, in CCM+GEN- than GEN-treated group and by 45% and 60%, respectively, in CCM+NaS- than NaS-treated group

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These observations underscore that interpreting ferroptosis mechanisms based on a single mediator is insufficient and may yield incomplete conclusions