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In addition, the enhancement of FAO can be mediated by G protein-coupled bile acid receptor TGR5 agonists and accompanied by an increase in mitochondrial superoxide dismutase 2 (SOD2) activity, which reduces oxidative stress and lipid accumulation in the podocytes thereby attenuating the DKD injury [70]

Nevertheless, formulation must be validated for (i) retention of biological activity after encapsulation, (ii) predictable release kinetics at the intended site, and (iii) avoidance of off-target thiol modification that could cause toxicity (El-Saadony et al., 2024

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Liver Mitochondrial Membrane Crosslinking and Destruction in a Rat Model of Wilson Disease

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