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Each of these effects is specific ( i.e ., NO-related) because when they were administered together, L-NAME and L-arginine regularly attenuated or antagonized each others responses[3,52,53]

doi:10.1002/smll.202107714 183

These results suggest that CYP2E1 plays an important role in ethanol-induced fatty liver disease and oxidative stress

reported that -GalCer administration activates NKT cells to produce IFN- and impede liver regeneration

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