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Paradoxically, this same mechanism may underlie its potential pro-cancer effects: by boosting GSH production, cancer cells can better protect themselves from oxidative damage caused by ROS (Ramachandran and Jaeschke, 2021)

In recent years, an increasing number of studies have focused on adjunctive therapeutic strategies aimed at further protecting dopaminergic neurons based on existing treatments, delaying disease progression, and improving patients quality of life

*Correspondence: Kun Chen, [email protected] This article was submitted to Cell Death and Survival, a section of the journal Frontiers in Cell and Developmental Biology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

In addition, SlBEL11 gene silencing resulted in changes in the expression of core enzyme genes in the carotenoid metabolic pathway, including PSY1 , P SY2 , PDS , ZDS , LCY-b1/b2 , CYC-b , and LCY-e

J Ardabil Univ Med Sci

Furthermore, patients with compensated liver disease exhibit a characteristic imbalance in the ratio between aromatic amino acids (AAAs) and branched-chain amino acids (BCAAs) [12]