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Comparative Analyses of Pentraxins: Implications for Protomer Assembly and Ligand Binding

identified the principal proteoglycan-binding site in LDL and showed that a single point mutation in apoB100 impaired binding to proteoglycans (231)

GLS1 functions as the primary isoform under most conditions, while GLS2 can be induced under specific contexts such as p53 activation, partially compensating for GLS1 activity and thereby adding complexity to its therapeutic targeting [183]

Branched-chain amino acids, especially of leucine and valine, mediate the protein restricted response in a piglet model

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