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Consistently, Rho0 cells, which lack functional ETC, were more sensitive to GPX4 inhibitionmediated mitochondrial lipid peroxidation and ferroptosis than control cells

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Perfusion bioreactors attenuated mitochondrial dysfunction and ER stress resulting in a favorable intracellular redox environment as indicated by improved reduced to oxidized glutathione ratio

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Conclusions Thalamic GSH reduction appears to differentiate migraine from TTH in this sample, supporting a distinct role of oxidative stress in migraine pathophysiology