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Carbon nanostructures: the drug and the delivery system for brain disorders

Jennifer Beatriz Silva Morais: (Wrote the paper)

Such behavioral deficits in A53T mice were accompanied by the presence of elevated -synuclein monomers and aggregates (Supplemental Figure 1J) and a remarkable loss of dopaminergic neurons in the substantia nigra, as indicated by tyrosine hydroxylase (TH) staining (Figure 1C)

These NPs demonstrate sustained release of dopamine and high cytocompatibility, showing potential in reducing oxidative cellular damage and providing neuroprotection

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