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10.1007/s12668-025-01911-9 86 JadhavK.DeoreS.DhamechaD.HrR.JagwaniS.JalalpureS.et al (2018)

[DOI] [Google Scholar] 264.Cunningham F.X., Jr., Gantt E

[DOI] [PMC free article] [PubMed] [Google Scholar] 140.Stolzenberg-Solomon R.Z., Chang S.C., Leitzmann M.F., Johnson K.A., Johnson C., Buys S.S., Hoover R.N., Ziegler R.G

The vesicular transport assays presented in this study provide indirect evidence that the Fe-DpC complex, in the presence of GSH, is a substrate of ABCC1

Accordingly, the therapeutic approach is to titratenot suppressthe UPR^ER, using chemical chaperones and selective PERK/IRE1 modulators to reduce misfolded-protein pressure while preserving adaptive signalling (Feldman et al

Beyond two specific interaction flags - Resveratrol with blood-thinning medications and Vitamin C with a history of kidney stones - no XenNAD+-specific adverse-event information was available in the materials reviewed