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Chlorogenic acid mitigates ferroptosis in macrophages induced by pneumolysin and streptococcus pneumoniae through activation of NRF2/GPX4 pathway
Researchers investigate how DSIP interacts with stress-response communication systems
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[88] Key mechanisms involved in this process include natural killer (NK) cell-mediated granule exocytosis, cytotoxicity mediated by CD8 + /CD4 + T cells, macrophage-driven efferocytosis and indirect clearance through dendritic cell regulation of T cells
