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Some studies have reported that -KG is closely related to histone methylation in cells, and histone methylation regulates cell growth

These have been reported to lead to impaired GH release (96) or to alter folding of GH (97)

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Other studies have revealed compositional and functional changes in gut microbiota (dysbiosis) and subclinical intestinal inflammation as possible drivers of TJ destabilization and leaky gut in animal models of arthritis and humans with RA [2, 3]