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At the same time, a previous paper has explained that the interaction between KEAP1 and NRF2 proteins can also be disrupted, affecting the stability of NRF2 and thereby promoting the further progression of breast cancer [117]

Peptides address biochemical repair

doi: 10.1128/AAC.46.6.1887-1895.2002 110 TsenovaLSokolKVictoriaHFKaplanG

These observations suggest that part of the survival propensity of C38 cells may be due to an enhanced capacity to continue to prevent lipid hydroperoxides formation (Fig

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This chronic activation impairs neuronal homeostasis, worsens synaptic dysfunction, and ultimately accelerates neurodegenerative processes [113116]