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Apoptosis is a process of programmed cell death that can contribute to pathological cell and tissue damage in cardiovascular and neurodegenerative diseases, and during the response to toxins (including chemotherapeutic agents), sepsis, or other environmental and physiological stimuli

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This series of pathological alterations further exacerbates local hypoxia and, through the classic hypoxia signaling pathway, stabilizes HIF-1, thereby driving disease progression [102]

Gain-of-function mutations in oncogenes (such as RAS superfamily) or positive regulators (such as NF-B inducing kinase) contribute to sustained activation of NF-B signaling and subsequent cell proliferation